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If you’ve ever witnessed someone suffer a stroke, you understand the humbling nature of this disease. It can reduce the mightiest human being to an immobile, helpless creature. Impairment of crucial functions like speech, walking, and control of bowel and bladder can wrench control from the body in a moment. Even perpetually youthful TV personality Dick Clark was struck down by stroke at age 75, despite the outward appearance of perfect health. Clark’s stroke resulted in a six-week hospital stay and, judging from fragmented reports, significant disability. Stroke can be like a devastating fire that strikes without warning, leaving only smoldering rubble. Stroke can so ravage basic bodily functions that often all you can hope for is to regain a portion through rehabilitation. The disease process that underlies stroke requires decades—30 or 40 years—to develop. With that much lead time, why aren’t we better able to detect or stop this crippling disease? The truth is that we are able to predict many, if not most, strokes. Advances in imaging technology allow detection of atherosclerotic plaque that cause stroke years before it becomes a threat. Progress in deciphering the causes of stroke has also leapt forward. Unfortunately, your neighborhood physician still focuses on diagnosing the crisis rather than anticipating it. Physicians prefer to deal with catastrophes and are just not that interested in prevention. Most physicians ask: “Is it time to operate or not?” The medical community obsesses over procedures like carotid endarterectomy (surgical removal of plaque) or carotid stents. Even when a person is afforded the warnings of a “mini-stroke”, or transient ischemic attack (TIA), little more is done once it’s determined that surgery is not necessary—even though this person has high risk for future stroke (50% over 10 years). Let’s flip-flop this approach to stroke. Procedures represent a failure of prevention! Where do strokes come from? Stroke develops when some portion of the brain is deprived of blood. This usually results from a tiny bit of debris that dislodges from an atherosclerotic plaque along the walls of an artery (the same sort that accumulates in coronaries causing heart attack). The sources of debris have been a subject of controversy, but new imaging technologies have settled the question. Any blood vessel that leads from the heart to the brain can be a source. The two carotid arteries on both sides of your neck are a frequent source, as these arteries are prone to develop plaque. (Our discussion will be confined to what are called thromboembolic, or ischemic, strokes, i.e, strokes that occur from plaque that fragments, sending debris to the brain, and will not include the far less common hemorrhagic strokes due to rupture of small vessels in the brain, nor will we discuss atrial fibrillation and other heart causes of stroke. The thromboembolic strokes we discuss cause around 88% of all strokes.) Over the last 10 years, the aorta has been recognized as another important source of stroke. The aorta is the main artery of the body whose branches go to the head, arms, and legs. Atherosclerotic plaque is a live tissue that, through poor diet, inactivity, high cholesterol, overweight, etc., grows and becomes progressively more unstable. At some point, plaque fragments. Little bits break away, traveling to the brain. Fractured plaque also exposes its deeper structures to flowing blood, triggering blood clot formation, which in turn can also fragment and go to the brain. Atherosclerotic plaque is a prerequisite for the most common causes of stroke. If the majority of strokes originate from plaque, why not measure plaque to determine if you’re at risk for stroke? How can we easily, safely, and accurately measure plaque in the carotid arteries and aorta? And if plaque can be measured, can it be shrunk or inactivated to reduce or eliminate risk for stroke? How can plaque be measured? Just 20 years ago, the only practical method of identifying plaque in the carotids or aorta was through angiography, requiring catheters inserted into the body to inject x-ray dye. Angiography was impractical as a screening measure. CT scanning and magnetic resonance imaging (MRI) are emerging as exciting methods of imaging both carotids and aorta. Unfortunately, most centers and physicians are much more focused on the diagnostic uses of these technologies for people who have already suffered stroke or other catastrophe, and application of these devices for preventive uses is still evolving. One exception is when aortic calcification or aortic enlargement is incidentally noted on the increasingly popular CT heart scans; this is an important finding that can signal presence of aortic plaque. The one test that is widely available and can be performed in just about any center is carotid ultrasound. It’s simple, painless, and precise. Two basic observations can be made: 1. Plaque detection—Atherosclerotic plaque can be clearly visualized. If plaque blocks more than 70% of the diameter of the vessel, or if there are “soft” (unstable) elements in plaque, then stroke risk may be high enough to justify surgery or stents. However, if there are plaques that are less severe, substantial risk for stroke may still be present that can be reduced with preventive measures. 2. Carotid intimal-medial thickness—This is a measure of the thickness of the lining of the carotid artery in areas not involved by plaque, but often precedes the development of mature plaque. Carotid intimal-medial thickness also provides an index of body-wide potential for atherosclerotic plaque that can place you at risk for stroke. The aorta, for instance, cannot be well imaged by surface ultrasound but can still be a source for stroke. Increased carotid intimal-medial thickness and carotid plaque are closely associated with likelihood of aortic plaque. The Rotterdam Study of 4000 participants demonstrated that if carotid intimal-medial thickness is greater than normal (1.0 mm), then you can be at risk for stroke (and heart attack), even if no carotid plaques are detected. Carotid ultrasound is the one test you should consider that provides the most information with least effort. Ultrasound is harmless, painless, and can be obtained just about anywhere. Even if your doctor disagrees with your request for a carotid ultrasound, an increasing number of mobile services are popping up nationwide that make this test available for around $100. One important point: many scanners and interpreters will only report whether plaque is present or not. While this is important information, you should request that the carotid-intimal medial thickness be made as well. Not all centers can make this simple measure (because of software requirements), but it doesn’t hurt to try. Any amount of carotid plaque is reason to follow a preventive program, even if the plaque is insufficient to justify surgery. Can plaque be reduced? Can we shrink plaque in carotid arteries and aorta and thereby reduce, perhaps eliminate, these sources of stroke? That question is gaining momentum as effective therapies become available that pack real punch for reducing plaque. Study after study has now documented that plaque can be reduced and, with it, risk for stroke. Reduction in plaque of 10–20% is possible within a year or two. Let’s consider the most potent influences on carotid and aortic plaque growth that need to be considered in a plaque-reducing program. (I assume that you are a non-smoker—if you are a smoker, you first need to concentrate on quitting.) Hypertension Considerable experience documents the power of blood pressure-lowering for prevention of stroke. The most recently updated guidelines, the JNC–VII, recommends a blood pressure of 407 mg/dl heightens stroke risk six-fold. C-reactive protein (CRP) This measure of inflammation is proving to be a useful marker for identifying people at risk for stroke, with increased risk beginning at a level of 0.5 mg/l. High CRP also predicts more rapidly growing carotid plaque. Homocysteine Homocysteine is an important marker of increased likelihood of both carotid and aortic plaque, as well as stroke. In 1997, the European Concerted Action Project reported more than a doubling of stroke when homocysteine levels exceeded 12 mol/l. As homocysteine increases to 20 μmol/l, risk for stroke and heart attack increases an amazing 10-fold over that at a level of 9 μmol/l. Asymmetric dimethylarginine (ADMA) ADMA is recently discovered amino acid whose blood levels can skyrocket up to 10-fold in the presence of hypertension, metabolic syndrome, diabetes, high cholesterol and triglycerides, obesity, and high homocysteine levels. ADMA blocks the action of the amino acid, l-arginine. This mimicry reduces the availability of nitric oxide, a powerful dilator and protector of arteries. ADMA levels in the top 10% predict a six-fold heightened risk for future stroke, and ADMA levels in people with strokes are double that in other people. A carotid ultrasound study in 116 subjects showed that higher blood levels of ADMA are associated with more severe carotid plaque. Because of ADMA’s shared role across a variety of abnormal conditions, correction or blocking the action of ADMA has been suggested as a unique therapeutic tool to reduce stroke risk. Cholesterol Data suggest that lowering cholesterol with statin cholesterol-lowering drugs slows carotid plaque growth and reduce stroke risk approximately 22%. An interesting study from the Cardiovascular Institute at Mt. Sinai School of Medicine in New York using the precise measuring ability of MRI of the carotids and thoracic aorta showed an impressive 20% regression of plaque area with simvastatin (Zocor®) taken for two years. Although guidelines for cholesterol treatment recommend reduction of LDL cholesterol to 100 mg/dl in high-risk persons, a report from the Walter Reed Army Medical Center in Washington, DC, showed that carotid plaque was more effectively reduced when LDL cholesterol of 70 mg/dl or lower was achieved with statin cholesterol drugs. Lower LDL cholesterol may, therefore, be better. Treatment Strategies to Reduce Carotid and Aortic Plaque The essential question: How do we reduce carotid and aortic plaque? If we make this the focus of our efforts, many pieces begin to fall into place. If you’ve had any measure of carotid or aortic plaque such as a carotid ultrasound or aortic calcification on a CT heart scan, you know that you’re at increased risk for stroke. You also have a baseline for future comparison to gauge whether your program is working or not. Because most people have not one but several causes of carotid and aortic plaque, there is no one single treatment that effectively eliminates risk for stroke. Instead, most people require a comprehensive program of healthy diet, exercise, supplements, and medication when indicated. Here, we focus on the nutritional supplements that can be critical components of your plaque-reduction program. Fish oil Fish oil is a cornerstone of your stroke prevention program. Epidemiological observations suggest a strong relationship of fish intake and reduction of stroke risk. Carotid ultrasound studies demonstrate less carotid plaque with greater intakes of fish. A cleverly designed University of Southampton study made the fascinating observation that fish oil transforms the structure of carotid plaque. 150 people with severe carotid plaque scheduled for carotid endarterectomy (surgical removal of the plaque) were given fish oil, sunflower oil, or no treatment over several months while waiting for their procedure. (Delays in the British health system permitted this unique design.) Plaque was removed at surgery and examined. Participants taking fish oil had reduced inflammation in plaque and thicker tissue covering the fatty core, markers of more stable plaque. Those taking sunflower oil or no treatment had unstable plaques with greater inflammation and thinner, less sturdy covering tissue. This suggests that fish oil stabilizes carotid plaque, making it less likely to rupture and fragment. A standard capsule of fish oil (containing 300 mg of EPA + DHA) contains the same amount of omega-3s as a 3 oz serving of cod or halibut; three capsules (900 mg DHA + EPA) contain the equivalent of a serving of farm-raised salmon. The dose that seems to provide greatest protection from stroke, lowers triglycerides (that form abnormal lipoproteins; see above), and reduces fibrinogen, is four capsules per day (1200 mg EPA + DHA). Coenzyme Q10 (CoQ10) Although there are no data specifically addressing whether CoQ10 reduces plaque, it is a marvelously effective way to reduce blood pressure, one of the crucial factors causing carotid and aortic plaque growth. A pooled analysis of eight studies showed that, on average, CoQ10 in daily doses of 50–200 mg reduced systolic blood pressure by 16 mm Hg, diastolic pressure by 10 mm Hg. Data suggest that CoQ10 can reverse abnormal heart muscle thickening (hypertrophy), another manifestation of high blood pressure, strongly suggesting that CoQ10 has benefits beyond just reducing pressure. Supplements to correct the metabolic syndrome Weight loss is, without question, the most immediate and direct path to correction of this dangerous pre-diabetic condition. A drop of even 10–20 lbs yields improvements across the board: increased sensitivity to insulin, increased HDL, and reductions in triglycerides, CRP, fibrinogen, small LDL particles, and blood pressure. Diet and exercise are fundamental components of an effort to lose weight; low carbohydrate or reduced glycemic index diets (e.g., South Beach or Mediterranean) rich in fibers are clearly effective. Several supplements can amplify weight-reduction efforts and be useful adjuncts to your lifestyle program. Among them: White bean extract White bean extract blocks intestinal absorption of carbohydrates by 66%. 1500 mg twice a day with meals yields, on average, 3–7 lbs of weight loss in the first month of use. The only side-effect is excessive gas, due to unabsorbed starches. Glucomannan This unique fiber taken prior to meals absorbs many times its weight in water and thereby fills your stomach. You consequently take in less food. Most people lose around four lbs per month using 1500 mg prior to each meal. Interestingly, glucomannan also blunts the rise in blood sugar after meals, an effect that, by itself, may lead to weight loss. Be sure to take with plenty of water. DHEA This adrenal hormone is key to maintaining physical stamina, mood, muscle mass in men, and libido in women. A recent randomized, placebo-controlled study at Washington University in 56 subjects showed a 13% decline in abdominal fat (fat that drives resistance to insulin) measured by MRI with 50 mg of DHEA per day at bedtime, along with improved sugar control and lower insulin levels. Pectin, beta-glucan Pectin is the soluble fiber in citrus rinds, green vegetables, and apples, also available as a supplement. Beta-glucan is the soluble fiber of oats and is also available as a supplement. Both are wonderful fibers that provide feelings of fullness, lower cholesterol, slow release of sugars, and can yield modest weight reduction. A USC study in 573 subjects using carotid ultrasound showed that greater intake of healthy fibers like pectin and beta-glucan is associated with less carotid plaque growth. Folic acid, vitamins B6 and B12 Dr. Daniel Hackam at the Stroke Prevention and Atherosclerosis Research Centre in Ontario conducted a study using carotid ultrasound in 101 participants treated with folic acid 2.5 mg, vitamin B6 25 mg, and B12 250 mcg per day. Treatment resulted in plaque reduction, especially when homocysteine levels exceeded 14μmol/l at the start, compared to untreated participants who experienced substantial plaque growth. An attempt to clarify the role of homocysteine treatment was made through a National Institute of Health-sponsored study of stroke prevention. 3680 participants with a prior history of stroke were enrolled and given either a “low-dose” (20 mcg folic acid, 0.2 mg B6, 6 mcg B12) or a “high-dose” (2.5 mg folic acid, 25 mg B6, 400 mcg B12) regimen. Although starting homocysteine levels showed a graded association with stroke risk (higher homocysteine levels predicted greater stroke risk), the treatment groups experienced, on average, only a 2 μmol drop in homocysteine levels and no reduction in stroke risk over two years. The study investigators as well as critics have suggested that the study failed due to an insufficient treatment period and that the doses were too low. (The doses we use in our plaque reduction program are folic acid 2.5–5.0 mg, B6 50–100 mg, B12 1000–2500 mcg.) L-arginine L-arginine can be used to overpower the adverse effects of ADMA. L-arginine is emerging as an important carotid plaque-reversing tool. Early reports in animals showed that l-arginine completely halted growth of aortic plaque, and did so more effectively than lovastatin (a cholesterol-lowering drug). In humans, L-arginine reduces blood pressure, abnormal constriction of carotid and coronary arteries, blocks entry of inflammatory cells into plaque, increases sensitivity to insulin, and heightens exercise capacity. Following coronary angioplasty or stent placement, l-arginine results in up to 36% reduction in plaque growth. The average American takes in 5400 mg of l-arginine through food every day. Supplementing with doses of 3000–12,000 mg per day has proven useful to correct many of these phenomena. (We use a dose of 6000 mg of l-arginine powder, twice a day on an empty stomach, dissolved in water, for our plaque regression program.) Does this result in a reduction of stroke risk? The emerging data suggest that l-arginine is likely to exert a powerful plaque-reducing and stroke-preventing benefit, but we await more clinical trial data. Conclusion Reducing stroke risk by reversing carotid and aortic plaque is becoming an everyday reality, with better tools becoming available. To know whether you’re at risk, the best and most available imaging tool is carotid ultrasound, aiming to identify intimal-medial thickness >1.0 mm, or carotid plaque. Any degree of calcification of the aorta, such as on a CT heart scan, is another useful measure of risk. Treatment to reduce risk is multi-faceted but is based on examining all your sources of risk, including metabolic syndrome, small LDL, lipoprotein(a), and C-reactive protein. Fish oil is the one absolutely crucial ingredient in any stroke prevention program. Other supplements can be used in a targeted fashion, depending on the causes identified for your carotid or aortic plaque. Ideally, repeat scanning of your carotids should be done sometime after your program has begun to assess whether you’ve successfully achieved reversal of plaque growth. penis elargement traction device enlargement manhattan pnis surgeon male penis enhancement free natural penile enlargement manual penis enlargment exercise cheap penile enlargment free penis enlarement technique penis enlargement tool

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There is nothing worse for a relationship than neglect, you need to keep your relationship fresh and passionate and this is what this article is all about. A relationship is like a bank account. If you constantly make withdrawals and no deposits, the bank account will soon close for lack of funds. So too, relying on a few plain old sexual positions will usually result in both partners of a relationship becoming bored, and eventually, the sexual (and love making) act, will appear bland and uninteresting. It seems that each couple develops a kind of routine; and if the couple really cares about each other, they will have a place in this routine for “new and cool stuff”. This article is about that, and it can keep your Sexlife alive and exciting. Consider a quick change from the bed in the bedroom is the chair in the living room. Believe it or not, having sex on a chair can be as varied as on the bed, gives the couple a whole new range of sensations, and can be fun as well. There are few basic positions, and these have also their variants, and allows the couple to be very creative (giving a warm loving feeling as well). Basic Position: Knelling on the Chair. In the position you use a standard dining room chair, with high back. The girl kneels on the chair and faces the back of the chair holding on to start. The man enters from the rear (a variant of the doggy-style pose) but with a lot of advantages. The guy can massage his partner, fondle her breasts, and stimulate her clitoris. This allows for deep penetration, and slow and long trusting is advised. The girl can push back with her hands creating greater friction. This is a very sexy position, and almost always ends up in mutual orgasm. Basic Position Two: Seeing “eye to eye” For this position you use either the sofa for an arm chair. The man sits down normally and the woman then sits on the man’s lap, but her legs over each of the arms of the chair. The man easily enters, and this position the couple are really eye to eye. The man now supports his partner at her waste, and helps her move up and down. This position allows for both maximum clitoral and G-spot stimulation and can end in a dramatic orgasm for the woman. Second Position: The Jackknife Here the couple are assumed to be more or less athletic. The woman rests her arms to the elbow on the chair seat, and holds on the back of the chair. The man then lifts her legs, and supports her, and the woman will wrap her legs around the man’s waist and back. The man enters her, and there is no trusting, only deep penetration. This has minimal clitoral or G-spot stimulation, but it is a very unique feeling and often the couple reach an orgasm quickly, as the movements are very intimate. Second Position: Across the Sea Here the woman lays across the arm chair, and not sitting on it. She is in a prone position, using one arm for a pillow and the other arm supporting her pelvic area. The man enters from the rear, and the woman then, if she wishes, can bring her legs together, giving the man an exquisite feeling. This allows for clitoral friction and some G-spot stimulation as the man’s penis will tend to trust downward. It is a wonderfully personal position, and allows for much intimacy between the couple. Keeping your Sexlife alive does require variety, so try the above. magna rx does penis enargement work vimax best penis enlargement does penis enhancement work penile enlargment picture cheap penile enlargment best penis enlargement pills penis enargement picture vimax male virility sexual enhancement

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Cosmetic surgery Costs – How to get world class treatment at lowest cost in India Recently the rise in medical tourism has seen cosmetic surgery costs drop by up to 90%, making world class treatments affordable for everyone and you also get to enjoy a free holiday as well! If you have always wanted a face lift, breast enlargement or another procedure you can now afford the look you have always dreamt about, as cosmetic surgery costs have tumbled. Huge cosmetic surgery cost savings An extensive facelift that would cost $20,000 in the U.S would cost under $3,000 in India saving a massive $17,000! Savings are massive across all procedures making cosmetic surgery within the reach of everyone. let’s look at how cosmetic surgery costs have been reduced and the treatments available. What Is Medical Tourism? This is where residents of one country seek to reduce the costs of their medical treatment at home, by having treatments done abroad and cutting the costs dramatically and enjoy a free holiday as well India The World Leader India combines world-class healthcare with prices costing a fraction of those in the US or Europe. This year around half a million foreign patients will travel to India for medical care, in 2002, the number was only 150,000. India is now the leader in medical tourism packages and growth in the industry is growing at more than 30% per annum and looks set to become a multi billion pound industry. Infestructure costs are far lower in India and this is reflected in lower cosmetic surgery costs, yet the hospitals are world class. The Expertise of Cosmetic Surgery in India There has been bad press on cheap cosmetic surgery operations and most of them have been in western countries! In India low cosmetic surgery costs are combined with surgery on par with anywhere in the world and this is one of the fastest areas of growth in medical tourism in India. Not only are the facilities fantastic, but Indian doctors are acknowledged worldwide for their expertise and professionalism, with many having trained in the west. The Price of Cosmetic Surgery in India Plastic and reconstructive surgery In India utilizes the latest techniques to cover all areas of cosmetic surgery including: • Hair restoration,hair implants, hair flaps, and scalp reductions • Face lifts - Rhytidectomy, Rhytidoplasty * Upper arm lift - Brachioplasty • Eyelid tuck, lower or upper - Blepharoplasty • Hair restoration, implants, flaps, and scalp reduction • Ear Surgery * Rhinoplasty nose jobs • Demabrasions • Laser hair removal . Otoplasty • Chin and cheek sculpting • Lip augmentation - Cheiloplasty * Cosmetic dentistry • breast surgery • Liposuction and tummy tuck These are just samples of cosmetic treatments available and there are many more and low cosmetic surgery costs are the norm in India not just a special deal and all treatments are on par with cosmetic surgery clinics elsewhere in the world. Conclusion So, you get cosmetic surgery costs at up to 90% lower than locally, world class treatments, doctors with unrivalled expertise and the chance to enjoy a relaxing break in one of the most beautiful and diverse countries on earth. From stunning beaches to the mighty Himalayas and thousands of years of culture and much more. For costs of all major treatments buy vigrx penile enlargement forum enlargment manhattan penis surgeon free exercise tip for penis elargement penile enlargment pic before and after penis elargement device penile enlargment patch penis enlagement pic vimax male virility sexual enhancement

A lot of men are ready to go to great lengths to get a bigger penis. Through the years, these men have tried any possible penis enlargement method, regardless of how ridiculous or dangerous that method was. Although there is no shortage of safe male enhancement techniques, some men seem hell-bent on putting into practice the strangest ideas and the most obscure penis enlargement techniques. One of the lesser known penis enlargement techniques is the injection of liquid silicone into the penis and the scrotum. This is a pretty drastic approach to penis enlargement and not frequently performed. The main advantage of this technique is a significant increase in girth. Silicone injections are not the best way of increasing the length of the penis shaft, but they do wonders for girth. The results are usually impressive. The biggest gains obtained through this method amounted to more than 900 percent increase in penis volume. However, it’s worth noting that a 900 percent increase in penis and scrotum volume renders the penis unusable for anything except urination. Moreover, the effects of silicone injections are, for better or for worse, irreversible. The injection of silicone into the penis has a high chance of disrupting tissue, blood vessels and nerves, causing loss of sensation and the inability to achieve penetration. If you think that losing the ability to have sex is too high a cost for a bigger penis, you are right. A silicone injection enlargement gone wrong turns the penis in a fashion accessory that cannot be used for sex anymore. Other side effects include a high chance of inflammation and discoloration of the surrounding tissue. The silicone also tends to cause the formation of granulomas, which are nodules of inflamed, granulated tissue. Silicone is also known to migrate as drops of this substance spread throughout the body from the initial pocket of injected silicone. For this reason, the FDA has refused to approve the injection of liquid silicone into the body. This penis enlargement method usually makes the penis look abnormal and also leaves scars behind. Silicone can be removed from the body, but the procedure is very difficult, especially if migration has already occurred. Silicone injection is a far too risky approach, especially when there are many other safer and easier ones on the market. Using pills, patches or traction devices is more comfortable and less risky than bringing a hazardous substance inside your body. When too many things can go wrong, it’s time to choose another path.